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“viral RNA accumulation”

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Observationobservedoriginal resultpositivemeasured

Claim · model-written summary

Viral RNA accumulation was low in resistant plants.

Taxon: riceOryza sativat0
Outcome: Accumulation of viral RNAe0
Direction: lowe0

Verbatim

e0
“Accumulation of viral RNA was low”
✓ located verbatim in the source · abstract, characters 1148–1181

t0
“Rice”
✓ located verbatim in the source · title, characters 17–21

Title

Near Immunity to Rice Tungro Spherical Virus Achieved

H. Huet; J. Wang; Elumalai Sivamani; et al. 1999

Other claims from this paper (10)

Observation More than 70% of transgenic plants carried one to five Rep gene copies.
Observation Rep integration occurred at one locus in most plants.
Intervention effect Antisense Rep plants had significant, moderate RTSV resistance of 60%.
Observation Antisense RNA accumulated substantially in antisense plants.
Intervention effect Full-length sense Rep plants resisted RTSV completely even with high inoculum.
Intervention effect Truncated sense Rep plants resisted RTSV completely even with high inoculum.
Mechanism The authors concluded Rep resistance was cosuppression rather than protein mediated.
Observation Resistance worked against geographically distinct RTSV isolates.
Observation RTSV resistant transgenic plants could not assist RTBV transmission.
Recommendation The authors proposed transgenic plants to limit tungro disease spread.

Intervention effectobservedoriginal resultpositivemeasured

Claim · model-written summary

At low temperatures, systemic viral RNA accumulation was suppressed.

Taxon: Vigna unguiculataVigna unguiculatat0
Outcome: viral RNA accumulatione0
Direction: suppressione0

Not reportedTreatmentComparator

Verbatim

e0
“in addition to viral RNA accumulation suppression at this site.”
✓ located verbatim in the source · abstract, characters 1541–1604

t0
“Vigna unguiculata”
✓ located verbatim in the source · title, characters 113–130

Title

Effect of Temperature on Symptoms Expression and Viral RNA Accumulation in Groundnut Bud Necrosis Virus Infected Vigna unguiculata

Ajeet Singh; Vipin Permar; A Basavaraj; et al. Iranian Journal of Biotechnology 2018. doi:10.21859/ijb.1846

Other claims from this paper (12)

Observation GBNV first caused chlorotic spots at the inoculation site, which became necrotic spots.
Observation Yellow mosaic symptoms appeared at the systemic site during GBNV infection.
Intervention effect At 30 and 25 °C, infected plants showed severe necrosis.
Intervention effect At 30 and 25 °C, viral RNA accumulation at the inoculation site was higher.
Intervention effect Higher temperatures facilitated systemic viral spread.
Observation Viral RNA accumulation was lower at systemic than inoculated sites.
Intervention effect At 20 and 15 °C, symptoms at the inoculation site decreased.
Intervention effect At 20 and 15 °C, viral RNA at the inoculation site decreased.
Observation At 15 °C, no systemic viral symptoms were seen.
Intervention effect GBNV infection caused greater H2O2 accumulation at 30 and 25 °C than at 20 and 15 °C.
Intervention effect GBNV induced more cell death at higher than lower temperatures.
Association The authors suggest viral RNA accumulation parallels H2O2 production and cell death in temperature-dependent GBNV infection.

Intervention effectobservedoriginal resultnegativemeasured

Claim · model-written summary

Movement protein increased viral RNA accumulation in transgenic tobacco leaves.

Taxon: tobaccoNicotiana tabacume0
Organ / stage: leavese0
Treatment: MPe0
Gene: MPe0
Outcome: viral RNA (vRNA) accumulatione0
Direction: increasede0

Not reportedComparator

Verbatim

e0
“the MP triggered necrosis response in transgenic tobacco leaves and increased the viral RNA (vRNA) accumulation.”
✓ located verbatim in the source · abstract, characters 418–530

Title

Orchid fleck dichorhavirus movement protein shows RNA silencing suppressor activity

Mikhail Oliveira Leastro; Vicente Pallás; Jesús Ángel Sánchez-Navarro Journal of General Virology 2022. doi:10.1099/jgv.0.001805

Other claims from this paper (9)

Intervention effect Orchid fleck virus movement protein triggered necrosis in transgenic tobacco leaves.
Intervention effect Movement protein expression increased potato virus X infection severity.
Intervention effect Movement protein expression increased potato virus X RNA accumulation.
Observation Movement protein showed no local RNA silencing suppressor activity in the turnip crinkle virus assay.
Observation The five OFV RNA1-encoded proteins showed no local RNA silencing suppressor activity in Nicotiana benthamiana.
Intervention effect Movement protein expression significantly reduced GFP-derived siRNA accumulation.
Intervention effect Movement protein inhibited long-distance spread of RNA silencing in 16c plants.
Intervention effect Movement protein did not affect short-distance spread of RNA silencing in 16c plants.
Mechanism The authors identify movement protein as an orchid fleck virus suppressor of systemic RNA silencing.

Observationobservedoriginal resultmeasured

Claim · model-written summary

Viral RNA accumulation was lower at systemic than inoculated sites.

Taxon: Vigna unguiculataVigna unguiculatat0
Organ / stage: systemic sitee0
Outcome: viral RNA accumulatione0
Direction: lesse0
Comparator: inoculated sitee0

Verbatim

e0
“However, viral RNA accumulation was less at the systemic site than the inoculated site.”
✓ located verbatim in the source · abstract, characters 1256–1343

t0
“Vigna unguiculata”
✓ located verbatim in the source · title, characters 113–130

Title

Effect of Temperature on Symptoms Expression and Viral RNA Accumulation in Groundnut Bud Necrosis Virus Infected Vigna unguiculata

Ajeet Singh; Vipin Permar; A Basavaraj; et al. Iranian Journal of Biotechnology 2018. doi:10.21859/ijb.1846

Other claims from this paper (12)

Observation GBNV first caused chlorotic spots at the inoculation site, which became necrotic spots.
Observation Yellow mosaic symptoms appeared at the systemic site during GBNV infection.
Intervention effect At 30 and 25 °C, infected plants showed severe necrosis.
Intervention effect At 30 and 25 °C, viral RNA accumulation at the inoculation site was higher.
Intervention effect Higher temperatures facilitated systemic viral spread.
Intervention effect At 20 and 15 °C, symptoms at the inoculation site decreased.
Intervention effect At 20 and 15 °C, viral RNA at the inoculation site decreased.
Observation At 15 °C, no systemic viral symptoms were seen.
Intervention effect At low temperatures, systemic viral RNA accumulation was suppressed.
Intervention effect GBNV infection caused greater H2O2 accumulation at 30 and 25 °C than at 20 and 15 °C.
Intervention effect GBNV induced more cell death at higher than lower temperatures.
Association The authors suggest viral RNA accumulation parallels H2O2 production and cell death in temperature-dependent GBNV infection.

Observationobservedoriginal resultpositivemeasured

Claim · model-written summary

CP was critical for viral RNA accumulation.

Taxon: plant virust0
Gene: CPe0
Outcome: viral RNA accumulatione0

Verbatim

e0
“CP is critical for viral RNA accumulation”
✓ located verbatim in the source · abstract, characters 866–907

t0
“plant virus”
✓ located verbatim in the source · title, characters 60–71

Title

From darkness to light: Genetic manipulation of an atypical plant virus unveils key insights into kitavirus biology, highlighting capsid protein and eIF4A engagement to drive viral infection

Mikhail Oliveira Leastro; Elliot Watanabe Kitajima; Vicente Pallas; et al. PLoS Pathogens 2025. doi:10.1371/journal.ppat.1013388

Other claims from this paper (15)

Method / resource A recombinant GFP-expressing CiLV-C was feasible for cilevirus research.
Observation Viral RNA1 was essential for replication and independently self-replicated.
Observation Viral RNA2 was crucial for movement.
Mechanism The intergenic region promoted CP gene expression.
Observation Movement protein p32 facilitated viral spread.
Intervention effect Deleting p61 did not affect virion assembly.
Observation The putative matrix protein p24 was critical for virion morphogenesis.
Intervention effect Deleting p15 attenuated disease symptoms.
Intervention effect Deleting p15-p61 attenuated symptoms.
Mechanism CP enhanced viral RNA accumulation independently of RSS.
Observation CP bound RNA and interacted with eIF4A.
Intervention effect Overexpressing eIF4A increased CiLV-C RNA accumulation.
Intervention effect Knockdown of eIF4A reduced viral RNA accumulation.
Observation Turnip crinkle virus showed similar results for eIF4A.
Intervention effect TCV CP restored RNA accumulation in CP-defective CiLV-C.

Intervention effectobservedoriginal resultno effectmeasured

Claim · model-written summary

AtOLP deficiency does not affect viral RNA accumulation.

Taxon: TuMVTurnip mosaic viruse1
Treatment: Deficiencye0
Interactor: ArabidopsisArabidopsise1host
Gene: AtOLPe0
Outcome: viral RNA accumulatione0
Direction: does not affecte0

Not reportedComparator

Verbatim

e0
“Deficiency or overexpression of AtOLP does not affect viral RNA accumulation.”
✓ located verbatim in the source · abstract, characters 836–913

e1
“TuMV-Arabidopsis pathosystem”
✓ located verbatim in the source · abstract, characters 531–559

Title

Turnip mosaic virus selectively subverts a PR‐5 thaumatin‐like, plasmodesmal protein to promote viral infection

Rongrong He; Yinzi Li; Mark A. Bernards; et al. New Phytologist 2024. doi:10.1111/nph.20233

Other claims from this paper (11)

Observation AtOSM34 is localized at plasmodesmata.
Observation AtOLP is localized at plasmodesmata.
Intervention effect TuMV infection upregulates AtOSM34 expression.
Intervention effect TuMV infection downregulates AtOLP expression.
Intervention effect AtOLP overexpression does not affect viral RNA accumulation.
Intervention effect AtOSM34 knockdown inhibits TuMV infection.
Intervention effect AtOSM34 overexpression promotes infection.
Mechanism AtOSM34 reduces plasmodesmal callose deposition to promote viral movement.
Mechanism AtOSM34 targets the viral replication complex to enhance replication.
Mechanism AtOSM34 suppresses the ROS-mediated antiviral response.
Mechanism The authors infer that TuMV selectively subverts AtOSM34 to aid infection.

Associationobservedoriginal resultmeasured

Claim · model-written summary

66K accumulation affects viral RNA replication in infected Arabidopsis cells.

Taxon: Arabidopsis thalianaArabidopsis thalianae0
Interactor: Turnip yellow mosaic virusTurnip yellow mosaic viruse1pathogen
Gene: 66Ke0
Outcome: viral RNA replicatione0

Not reportedDirection

Verbatim

e0
“accumulation level of 66K affects viral RNA replication in infected Arabidopsis thaliana cells”
✓ located verbatim in the source · abstract, characters 408–502

e1
“Turnip yellow mosaic virus (TYMV) RdRp”
✓ located verbatim in the source · abstract, characters 270–308

Title

The Ubiquitin-Proteasome System Regulates the Accumulation ofTurnip yellow mosaic virusRNA-Dependent RNA Polymerase during Viral Infection

Laurent Camborde; Séverine Planchais; Vincent Tournier; et al. The Plant Cell 2010. doi:10.1105/tpc.109.072090

Other claims from this paper (6)

Observation TYMV polymerase is degraded by proteasome late in infection.
Observation The N-terminal noncatalytic domain contains degradation determinants.
Observation 66K is not a natural N-end rule substrate.
Mechanism A proposed 66K PEST sequence transfers degradation activity.
Observation Several Lys ubiquitylation target residues were mapped.
Intervention effect Mutation of these Lys residues stabilized 66K.

Intervention effectobservedoriginal resultno effectmeasured

Claim · model-written summary

AtOLP overexpression does not affect viral RNA accumulation.

Taxon: TuMVTurnip mosaic viruse1
Treatment: overexpression of AtOLPe0
Interactor: ArabidopsisArabidopsise1host
Gene: AtOLPe0
Outcome: viral RNA accumulatione0
Direction: does not affecte0

Not reportedComparator

Verbatim

e0
“Deficiency or overexpression of AtOLP does not affect viral RNA accumulation.”
✓ located verbatim in the source · abstract, characters 836–913

e1
“TuMV-Arabidopsis pathosystem”
✓ located verbatim in the source · abstract, characters 531–559

Title

Turnip mosaic virus selectively subverts a PR‐5 thaumatin‐like, plasmodesmal protein to promote viral infection

Rongrong He; Yinzi Li; Mark A. Bernards; et al. New Phytologist 2024. doi:10.1111/nph.20233

Other claims from this paper (11)

Observation AtOSM34 is localized at plasmodesmata.
Observation AtOLP is localized at plasmodesmata.
Intervention effect TuMV infection upregulates AtOSM34 expression.
Intervention effect TuMV infection downregulates AtOLP expression.
Intervention effect AtOLP deficiency does not affect viral RNA accumulation.
Intervention effect AtOSM34 knockdown inhibits TuMV infection.
Intervention effect AtOSM34 overexpression promotes infection.
Mechanism AtOSM34 reduces plasmodesmal callose deposition to promote viral movement.
Mechanism AtOSM34 targets the viral replication complex to enhance replication.
Mechanism AtOSM34 suppresses the ROS-mediated antiviral response.
Mechanism The authors infer that TuMV selectively subverts AtOSM34 to aid infection.

Associationobservedoriginal resultmeasured

Claim · model-written summary

Viral RNA accumulation corroborated symptom severity.

Taxon: watermelonCitrullus lanatuse1
Interactor: CGMMVCucumber green mottle mosaic viruse2pathogen
Outcome: severity of the symptomse0
Qualifier: accumulation of viral RNAe0

Not reportedDirection

Verbatim

e0
“the accumulation of viral RNA corroborated the severity of the symptoms”
✓ located verbatim in the source · abstract, characters 2011–2082

e1
“watermelon plants”
✓ located verbatim in the source · abstract, characters 1579–1596

e2
“CGMMV infection”
✓ located verbatim in the source · abstract, characters 1629–1644

Title

The 96th Amino Acid of the Coat Protein of Cucumber Green Mottle Mosaic Virus Affects Virus Infectivity

Zhenwei Zhang; Liming Liu; Huijie Wu; et al. Viruses 2017. doi:10.3390/v10010006

Other claims from this paper (7)

Intervention effectobservedoriginal resultnegativemeasured

Claim · model-written summary

NbPLDα1 loss of function promoted viral RNA accumulation.

Taxon: Nicotiana benthamianaNicotiana benthamianat0
Treatment: Loss of function of NbPLDα1e0
Gene: NbPLDα1e0
Outcome: viral RNA accumulatione0
Direction: promotede0

Not reportedComparator

Verbatim

e0
“Loss of function of NbPLDα1 inhibited virus-induced activation of MAPK cascades and promoted viral RNA accumulation”
✓ located verbatim in the source · abstract, characters 1285–1400

t0
“Nicotiana benthamiana”
✓ located verbatim in the source · abstract, characters 438–459

Title

Activation of MAPK-mediated immunity by phosphatidic acid in response to positive-strand RNA viruses

Jiayu Lin; Jinpeng Zhao; Linlin Du; et al. Plant Communications 2023. doi:10.1016/j.xplc.2023.100659

Other claims from this paper (14)

Observation Phosphatidic acid responded early to PVY infection.
Mechanism NbPLDα1 was responsible for higher PA during PVY infection.
Observation NbPLDα1 played an antiviral role.
Mechanism PVY 6K2 interacted with NbPLDα1 and raised PA levels.
Mechanism PVY 6K2 recruited NbPLDα1 and PA to viral replication complexes.
Mechanism PVY 6K2 activated MAPK signaling through NbPLDα1 and PA.
Mechanism PA bound WIPK/SIPK/NTF4 and prompted WRKY8 phosphorylation.
Intervention effect Exogenous PA spray activated the MAPK pathway.
Intervention effect Knockdown of the MEK2-WIPK/SIPK-WRKY8 cascade enhanced PVY genomic RNA accumulation.
Observation Turnip mosaic virus 6K2 interacted with NbPLDα1.
Observation Tomato bushy stunt virus p33 interacted with NbPLDα1.
Observation Turnip mosaic virus 6K2 induced MAPK-mediated immunity.
Observation Tomato bushy stunt virus p33 induced MAPK-mediated immunity.
Intervention effect NbPLDα1 loss of function inhibited virus-induced MAPK activation.

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“viral RNA accumulation”

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