By interacting organism as written › B . cinerea

Mechanismauthor interpretationoriginal resultmeasured

Claim · model-written summary

The authors infer that smooth-wound resistance does not depend on major disease signaling pathways.

Taxon: tomatoSolanum lycopersicumt0
Interactor: B . cinereae0pathogen
Outcome: absolute resistancee0
Qualifier: major disease signalling pathwayse0

Verbatim

e0
“this remarkable absolute resistance to B . cinerea does not depend on the major disease signalling pathways”
✓ located verbatim in the source · abstract, characters 1559–1666

t0
“tomato”
✓ located verbatim in the source · title, characters 70–76

Title

Resistance against B otrytis cinerea in smooth leaf pruning wounds of tomato does not depend on major disease signalling pathways

T. Beyers; C. Vos; R. Aerts; et al. Plant Pathology 2013. doi:10.1111/ppa.12075

Other claims from this paper (9)

Intervention effect Flush pruning gave tomato stem wounds absolute resistance.
Observation Petiole stubs were highly susceptible to B. cinerea.
Observation Wound resistance became nearly complete within 48 hours after deleafing.
Mechanism Wound drying did not explain the decline in susceptibility.
Association Disease-signaling mutants showed altered susceptibility to B. cinerea.
Mechanism Ethylene signaling was implicated in petiole-stub defense.
Association Jasmonate-biosynthesis mutants had decreased susceptibility.
Association Abscisic-acid-biosynthesis mutants had decreased susceptibility.
Association Smooth-stem wound resistance persisted despite salicylic acid, ethylene, jasmonate or abscisic acid signaling disruption.

Intervention effectobservedoriginal resultpositivemeasured

Claim · model-written summary

In OsBIANK1-overexpressing plants, expression of defence genes PR-1, PR-2 and PDF1.2 was up-regulated after infection with B. cinerea or P. syringae pv. tomato DC3000.

Taxon: ArabidopsisArabidopsist0
Treatment: O s BIANK 1 ‐overexpressinge0
Interactor: B . cinereaBotrytis cinereae0pathogen
Second interactor: P . syringae pv. tomato DC 3000Pseudomonas syringae pv. tomatoe0pathogen
Gene: PR ‐ 1 , PR ‐ 2 and PDF 1.2e0
Outcome: expression of some of well‐known defence genese0
Direction: up‐regulatede0

Not reportedComparator

Verbatim

t0
“Arabidopsis”
✓ located verbatim in the source · title, characters 87–98

e0
“In O s BIANK 1 ‐overexpressing plants, expression of some of well‐known defence genes (e.g. PR ‐ 1 , PR ‐ 2 and PDF 1.2 ) was up‐regulated after infection with B . cinerea or P . syringae pv. tomato DC 3000”
✓ located verbatim in the source · abstract, characters 912–1118

Title

Ectopic Expression of Rice OsBIANK1, Encoding an Ankyrin Repeat‐Containing Protein, in Arabidopsis Confers Enhanced Disease Resistance to Botrytis cinerea and Pseudomonas syringae

Dayong Li; Fengrang Wang; Bo Liu; et al. Journal of Phytopathology 2012. doi:10.1111/jph.12023

Other claims from this paper (4)

Intervention effectobservedoriginal resultpositivemeasured

Claim · model-written summary

Heat alone reduced B. cinerea decay by about 30%.

Treatment: heat treatment alonee0
Interactor: B . cinereae0pathogen
Outcome: decay caused by B . cinereae0
Direction: reducede0
Value: about 30%e0

Not reportedTaxonComparator

Verbatim

e0
“The heat treatment alone reduced decay caused by B . cinerea by about 30%”
✓ located verbatim in the source · abstract, characters 271–344

Title

EFFECTS OF COMBINING POSTHARVEST CALCIUM AND HEAT TREATMENT ON REDUCING DECAY AND MAINTAINING QUALITY IN APPLES

William S Conway; Carl E. Sams; Chien Yi Wang; et al. HortScience 1993. doi:10.21273/hortsci.28.5.587c

Other claims from this paper (7)

Intervention effect Heat with 2% CaCl 2 reduced decay by about 60%.
Intervention effect 2% CaCl 2 alone reduced decay by 40%.
Intervention effect 4% CaCl 2 alone reduced decay by 60%.
Intervention effect Heat alone or with calcium best maintained firmness at 80 N.
Intervention effect Calcium alone maintained firmness at 70 N, compared with 66 N in controls.
Observation Instron test curves distinguished heated from nonheated fruit qualitatively and quantitatively.
Intervention effect Heat after calcium infiltration increased surface injury compared with either treatment alone.

Observationobservedoriginal resultnegativemeasured

Claim · model-written summary

Fruit-isolated Botrytis cinerea was resistant to captan in vitro.

Taxon: black raspberryRubus occidentalise1
Interactor: B . cinereaBotrytis cinereae0pathogen
Outcome: resistance to captane0

Not reportedDirection

Verbatim

e0
“B . cinerea isolated from the fruit had developed resistance to captan”
✓ located verbatim in the source · abstract, characters 854–924

e1
“black raspberry”
✓ located verbatim in the source · abstract, characters 718–733

Title

FRUIT ROT CONTROL IN SMALL FRUIT CROPS WITH A NATURALLY-PRODUCED CHEMICAL

Fumiomi Takeda; Wojciech Janisiewicz HortScience 1990. doi:10.21273/hortsci.25.6.625a

Other claims from this paper (7)

Intervention effect Field application of Pseudomonas cepacia had variable control of Botrytis fruit rot in strawberry.
Intervention effect Pyrrolnitrin was as effective as Benlate/captan sprays in field applications.
Intervention effect Postharvest 100 ppm pyrrolnitrin dip inhibited pathogen growth for three days at room temperature.
Intervention effect A pyrrolnitrin dip plus cold storage extended fruit shelf life another five days.
Intervention effect Preharvest pyrrolnitrin sprays delayed rot in Bristol black raspberry by 4 to 5 days.
Intervention effect Captan treatment provided no rot protection.
Recommendation Authors suggest naturally produced compounds could reduce postharvest rots without synthetic fungicides.

Associationobservedoriginal resultmeasured

Claim · model-written summary

Flower susceptibility to B. cinerea was inversely correlated with mean vapor pressure deficit, r = 0.98.

Taxon: Rosa hybridaRosa hybrid cultivart0
Interactor: B . cinereaBotrytis cinereae0pathogen
Outcome: Susceptibility of the flowers to B . cinereae0
Direction: inverselye0
Value: r = 0.98e0
Qualifier: overall mean vapor pressure deficit from 0800 to 1900 hr for the 5-week growth period before harveste0

Verbatim

e0
“Susceptibility of the flowers to B . cinerea was correlated linearly ( r = 0.98) and inversely to the overall mean vapor pressure deficit from 0800 to 1900 hr for the 5-week growth period before harvest”
✓ located verbatim in the source · abstract, characters 923–1125

t0
“Quantification of the Impact of Environment on the Susceptibility of Rosa hybrida Flowers to Botrytis cinerea”
✓ located verbatim in the source · title, characters 0–109

Title

Quantification of the Impact of Environment on the Susceptibility of Rosa hybrida Flowers to Botrytis cinerea

James J. Marois; Judith C. Redmond; James D. MacDonald Journal of the American Society for Horticultural Science 1988. doi:10.21273/jashs.113.6.842

Other claims from this paper (3)

Recommendationauthor interpretationoriginal resultbasis n/a

Claim · model-written summary

Reduced dicarboximide use is recommended to avoid resistant Botrytis strains.

Taxon: boysenberriest0
Interactor: B . cinereaBotrytis cinereae0pathogen
Outcome: reduced use of dicarboximidese0
Setting: New Zealande0

Verbatim

e0
“a reduced use of dicarboximides is recommended to avoid problems associated with the resistant strains of B . cinerea identified in New Zealand.”
✓ located verbatim in the source · abstract, characters 688–832

t0
“Boysenberries”
✓ located verbatim in the source · title, characters 0–13

Title

Boysenberries: Control of botrytis fruit rot by fungicides

L. H. Cheah; K. G. Tate; P. T. Holland New Zealand Journal of Crop and Horticultural Science 1987. doi:10.1080/03015521.1987.10425535

Other claims from this paper (8)

Intervention effect All tested fungicides controlled botrytis fruit rot.
Intervention effect Dicarboximides and dichlofluanid were more effective than captan, thiram or folpet.
Intervention effect The reduced dicarboximide schedule yielded slightly more berry rot than the full schedule.
Intervention effect The reduced schedule did not reduce marketable yield.
Observation Residue decay half-life for iprodione was 10–12 days.
Observation Residue decay half-life for procymidone was 9 days.
Observation Residue decay half-life for vinclozolin was 7 days.
Observation Residue decay half-life for myclozolin was 5.5 days.

B . cinerea 36 claims

36 claims