Claim · model-written summary
Artificial microRNA downregulation of SOC3 rescued chs1-2 chilling sensitivity and defense defects.
- Taxon
- Arabidopsis
Arabidopsist0 - Treatment
- downregulation of SOC3 by an artificial microRNA constructe0
- Gene
- SOC3e0
- Outcome
- chilling sensitivity and defense defectse0
- Direction
- fully rescuede0
- Comparator
- chs1-2e0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“downregulation of SOC3 by an artificial microRNA construct fully rescued the chilling sensitivity and defense defects of chs1-2.”
t0“Arabidopsis genome”
Title
Temperature‐dependent autoimmunity mediated by chs1 requires its neighboring TNL gene SOC3
Yao Zhang; Yuancong Wang; Jingyan Liu; et al. New Phytologist 2016. doi:10.1111/nph.14216
Other claims from this paper (6)
- Observation Independent chs1 suppressor mutants carried mutations in SOC3 adjacent to CHS1.
- Observation SOC3 expression was upregulated in the chs1-2 mutant.
- Intervention effect Mutations in six soc3 alleles rescued chs1-2 chilling sensitivity and defense defects.
- Observation CHS1 interacted with the NB and LRR domains of SOC3.
- Observation Mutated chs1 interacted with TIR, NB and LRR domains of SOC3 in vitro and in vivo.
- Mechanism The authors conclude that CHS1-SOC3 interaction modulates temperature-dependent autoimmunity.