Claim · model-written summary
Salinity strongly promoted sucrose, citrate and malate accumulation in red fruit.
- Taxon
- tomato
Solanum lycopersicumt0 - Organ / stage
- red fruite0
- Treatment
- Salinity stresse0
- Outcome
- accumulation of sucrose, citrate, malatee0
- Direction
- strongly promotede0
Not reportedMaterialGeneCompoundInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Salinity stress strongly promoted the accumulation of sucrose, citrate, malate, and glutamate, and slightly promoted glucose and γ-amino butyric acid in red fruit.”
t0“Tomato”
Title
Takeshi Saito; Chiaki Matsukura; Yusuke Ban; et al. Journal of the Japanese Society for Horticultural Science 2008. doi:10.2503/jjshs1.77.61
Other claims from this paper (9)
- Intervention effect Salinity significantly enhanced Brix, surface color density and flesh firmness.
- Intervention effect Salinity suppressed fruit enlargement.
- Intervention effect Salinity strongly promoted glutamate accumulation in red fruit.
- Intervention effect Salinity slightly promoted glucose and γ-amino butyric acid in red fruit.
- Observation Ethylene-synthetic 1-aminocyclopropane-1-carboxylate oxidase was up-regulated earlier in stressed fruit.
- Observation Cab-1B was down-regulated earlier in stressed fruit.
- Intervention effect Phytoene synthase 1 was up-regulated under moderate salinity at the red stage.
- Intervention effect PEPCK was up-regulated under moderate salinity at the red stage.
- Mechanism PEPCK up-regulation suggested gluconeogenesis contributes to sugar accumulation in salinity-stressed fruit.