Claim · model-written summary
Salicylic acid supplied to AIP-treated plants restored resistance.
- Taxon
- Arabidopsis
Arabidopsist0 - Treatment
- Salicylic acid (SA), supplied to AIP-treated plantse0
- Outcome
- resistancee0
- Direction
- restorede0
- Comparator
- AIP-treated plantse0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“Resistance of Arabidopsis to Peronospora parasitica”
e0“Salicylic acid (SA), supplied to AIP-treated plants, restored resistance”
Title
B. Mauch-Mani; A. J. Slusarenko The Plant Cell 1996. doi:10.1105/tpc.8.2.203
Other claims from this paper (12)
- Observation PAL1 promoter activity was induced only near fungal structures in the incompatible interaction.
- Observation The PAL1 promoter was activated in cells undergoing lignification in the incompatible interaction.
- Intervention effect AIP pretreatment made Arabidopsis seedlings completely susceptible to Peronospora parasitica.
- Intervention effect AIP treatment suppressed lignification in Arabidopsis.
- Intervention effect AIP treatment prevented detection of pathogen-induced PAL1 promoter activity.
- Intervention effect OH-PAS caused a shift toward susceptibility in Arabidopsis, less pronounced than AIP.
- Intervention effect OH-PAS suppressed pathogen-induced lignification in Arabidopsis.
- Intervention effect OH-PAS did not suppress pathogen-induced PAL1 promoter activation.
- Intervention effect Endogenous salicylic acid levels increased significantly in the incompatible but not in the compatible combination.
- Intervention effect The increase in endogenous SA was suppressed by AIP but not by OH-PAS.
- Mechanism The results give evidence of a central role of SA in genetically determined plant disease resistance.
- Mechanism Lignification per se is not the deciding factor between resistance and susceptibility, although it is a component of the resistance mechanism.