Claim · model-written summary
CPK6 disruption impaired methyl jasmonate induced stomatal closure.
- Taxon
- Arabidopsis
Arabidopsist0 - Treatment
- Disruption of the CPK6 genee0
- Gene
- CPK6e0
- Outcome
- MeJA-induced stomatal closuree0
- Direction
- impairede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Disruption of the CPK6 gene impaired MeJA-induced stomatal closure”
t0“Arabidopsis”
Title
Shintaro Munemasa; Mohammad Anowar Hossain; Yoshimasa Nakamura; et al. PLANT PHYSIOLOGY 2010. doi:10.1104/pp.110.162750
Other claims from this paper (7)
- Intervention effect Disruption of the other CDPK genes did not impair methyl jasmonate induced closure.
- Observation No other marked methyl jasmonate insensitive phenotype was found in cpk6-1.
- Observation Methyl jasmonate activation of calcium permeable cation channels was impaired in cpk6-1.
- Observation Methyl jasmonate induced cytosolic calcium increments were reduced in cpk6-1.
- Observation Methyl jasmonate failed to activate slow anion channels in cpk6-1 guard cells.
- Observation Methyl jasmonate elicited reactive oxygen species and nitric oxide in cpk6-1 as in wild type.
- Mechanism CPK6 functions as a positive regulator of methyl jasmonate signaling in guard cells.