Oryza sativa 577,275 claims

species · family Poaceae · green plant · NCBI Taxonomy ↗

Common name: rice — Also written in claims: rice · upland rice · rice plants · hybrid rice · japonica rice · rice plant

Listed here with its subspecies, varieties and cultivar groups: Oryza sativa Japonica Group (5,865 claims), Oryza sativa Indica Group (3,837 claims), Oryza sativa f. spontanea (46 claims)

1687 spellings

Questions studied on this organism5,183 questions240 with results that differ25,631 papers

Oryza sativa · picture from its Wikimedia Commons categoryDesmanthus4food · CC BY-SA 3.0 us · Wikimedia Commons ↗

577,275 claims

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Observationobservedoriginal resultmeasured

Claim · model-written summary

Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.

Taxon: riceOryza sativat0
Treatment: iron deficiency or OsHRZ knockdowne0
Gene: OsbZIP83e0
Outcome: differential regulation between the transcript and protein levelse0

Verbatim

t0
“rice”
✓ located verbatim in the source · title, characters 106–110

e0
“In response to iron deficiency or OsHRZ knockdown,many genes showed differential regulation between the transcript and protein levels,including the TGA-type basic leucine zipper transcription factor OsbZIP83”
✓ located verbatim in the source · abstract, characters 413–620

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Observationobservedoriginal resultmeasured

Claim · model-written summary

OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.

Taxon: riceOryza sativat0
Gene: OsGRX6e0
Outcome: OsHRZ-interacting proteinse0
Setting: yeast and plant cellse0

Verbatim

t0
“rice”
✓ located verbatim in the source · title, characters 106–110

e0
“two glutaredoxins,OsGRX6 and OsGRX9, as OsHRZ-interacting proteins in yeast and plant cells”
✓ located verbatim in the source · abstract, characters 641–732

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Observationobservedoriginal resultmeasured

Claim · model-written summary

OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.

Taxon: riceOryza sativat0
Gene: OsGRX9e0
Outcome: OsHRZ-interacting proteinse0
Setting: yeast and plant cellse0

Verbatim

t0
“rice”
✓ located verbatim in the source · title, characters 106–110

e0
“two glutaredoxins,OsGRX6 and OsGRX9, as OsHRZ-interacting proteins in yeast and plant cells”
✓ located verbatim in the source · abstract, characters 641–732

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Observationobservedoriginal resultmeasured

Claim · model-written summary

OsGRX6 interacted with OsbZIP83.

Taxon: riceOryza sativat0
Gene: OsGRX6e0
Outcome: interacted with OsbZIP83e0

Verbatim

t0
“rice”
✓ located verbatim in the source · title, characters 106–110

e0
“OsGRX6 also interacted with OsbZIP83”
✓ located verbatim in the source · abstract, characters 734–770

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Observationauthor interpretationoriginal resultmeasured

Claim · model-written summary

In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.

Taxon: riceOryza sativat0
Gene: OsbZIP83e0
Outcome: 26S proteasome- and OsHRZ-dependent degradatione0

Verbatim

t0
“rice”
✓ located verbatim in the source · title, characters 106–110

e0
“OsbZIP83,OsGRX6 and OsGRX9 proteins are subjected to 26S proteasome- and OsHRZ-dependent degradation”
✓ located verbatim in the source · abstract, characters 814–914

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Observationauthor interpretationoriginal resultmeasured

Claim · model-written summary

In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.

Taxon: riceOryza sativat0
Gene: OsGRX6e0
Outcome: 26S proteasome- and OsHRZ-dependent degradatione0

Verbatim

t0
“rice”
✓ located verbatim in the source · title, characters 106–110

e0
“OsbZIP83,OsGRX6 and OsGRX9 proteins are subjected to 26S proteasome- and OsHRZ-dependent degradation”
✓ located verbatim in the source · abstract, characters 814–914

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Observationauthor interpretationoriginal resultmeasured

Claim · model-written summary

In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.

Taxon: riceOryza sativat0
Gene: OsGRX9e0
Outcome: 26S proteasome- and OsHRZ-dependent degradatione0

Verbatim

t0
“rice”
✓ located verbatim in the source · title, characters 106–110

e0
“OsbZIP83,OsGRX6 and OsGRX9 proteins are subjected to 26S proteasome- and OsHRZ-dependent degradation”
✓ located verbatim in the source · abstract, characters 814–914

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Observationauthor interpretationoriginal resultmeasured

Claim · model-written summary

OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.

Taxon: riceOryza sativae0
Organ / stage: rootse0
Gene: OsbZIP83e0
Outcome: protein was preferentially degradede0
Setting: iron-deficient conditionse0

Verbatim

e0
“OsbZIP83 protein was preferentially degraded under iron-deficient conditions in rice roots”
✓ located verbatim in the source · abstract, characters 985–1075

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Intervention effectobservedoriginal resultpositivemeasured

Claim · model-written summary

OsGRX9 overexpression improved rice tolerance to iron deficiency.

Taxon: riceOryza sativae0
Treatment: overexpressing OsGRX9e0
Gene: OsGRX9e0
Outcome: tolerance to iron deficiencye0
Direction: improvede0

Not reportedComparator

Verbatim

e0
“Transgenic rice lines overexpressing OsGRX9 and OsbZIP83 showed improved tolerance to iron deficiency”
✓ located verbatim in the source · abstract, characters 1077–1178

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsbZIP83 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.

Intervention effectobservedoriginal resultpositivemeasured

Claim · model-written summary

OsbZIP83 overexpression improved rice tolerance to iron deficiency.

Taxon: riceOryza sativae0
Treatment: overexpressing OsGRX9 and OsbZIP83e0
Gene: OsbZIP83e0
Outcome: tolerance to iron deficiencye0
Direction: improvede0

Not reportedComparator

Verbatim

e0
“Transgenic rice lines overexpressing OsGRX9 and OsbZIP83 showed improved tolerance to iron deficiency”
✓ located verbatim in the source · abstract, characters 1077–1178

Title

The basic leucine zipper transcription factor OsbZIP83 and the glutaredoxins OsGRX6 and OsGRX9 facilitate rice iron utilization under the control of OsHRZ ubiquitin ligases

KOBAYASHI,Takanori; SHINKAWA,Haruka; NAGANO,Atsushi J; et al. Institutional Repositories DataBase (IRDB)

Other claims from this paper (14)

Observation Iron deficiency or OsHRZ knockdown produced differential regulation between transcript and protein levels for many genes, including OsbZIP83.
Observation OsGRX6 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX9 interacted with OsHRZ proteins in yeast and plant cells.
Observation OsGRX6 interacted with OsbZIP83.
Observation In vitro assays suggested OsbZIP83 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX6 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation In vitro assays suggested OsGRX9 undergoes 26S proteasome- and OsHRZ-dependent degradation.
Observation OsbZIP83 protein was preferentially degraded in iron-deficient rice roots.
Intervention effect OsGRX9 overexpression improved rice tolerance to iron deficiency.
Observation OsGRX9 knockdown affected expression of iron-related genes.
Observation OsGRX6 knockdown affected expression of iron-related genes.
Intervention effect OsbZIP83 overexpression enhanced expression of OsYSL2 and OsNAS3.
Intervention effect OsbZIP83 overexpression enhanced expression of OsGRX9 and genes related to phytoalexin biosynthesis and the salicylic acid pathway.
Mechanism The authors infer that OsbZIP83, OsGRX6 and OsGRX9 facilitate iron utilization downstream of OsHRZ.