Claim · model-written summary
Endodermal ABA insensitive1-1 misexpression substantially restored lateral-root growth under salt.
- Taxon
- Arabidopsis
Arabidopsist0 - Organ / stage
- endodermise0
- Treatment
- misexpression of the ABA insensitive1-1 mutant proteine0
- Gene
- ABA insensitive1-1 mutant proteine0
- Outcome
- LR growthe0
- Direction
- recoverye0
Not reportedMaterialCompoundInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“In the endodermis, misexpression of the ABA insensitive1-1 mutant protein, which dominantly inhibits ABA signaling, leads to a substantial recovery in LR growth under salt stress conditions.”
t0“Arabidopsis”
Title
Endodermal ABA Signaling Promotes Lateral Root Quiescence during Salt Stress inArabidopsisSeedlings
Lina Duan; Daniela Dietrich; Chong Han Ng; et al. The Plant Cell 2013. doi:10.1105/tpc.112.107227
Other claims from this paper (5)
- Intervention effect Salt stress extended postemergence lateral-root quiescence and suppressed growth for days before recovery.
- Association Lateral-root quiescence correlated with sustained ABA response.
- Mechanism Quiescence depended on ABA biosynthesis, signaling, and transcriptional regulation genes.
- Mechanism Gibberellic acid signaling antagonized ABA and acted primarily in the endodermis.
- Mechanism Authors identify an endodermal ABA guard preventing root growth into saline environments.