Claim · model-written summary
Silencing 9-LOX, 13-LOX or α-DOX-1 attenuated the cell-death symptoms caused by PVX-PVY or TSWV infection.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianae5 - Treatment
- Silencing either 9-lipoxygenase (LOX), 13-LOX, or α-dioxygenase-1 (α-DOX-1)e0
- Interactor
- PVX-PVYe0pathogen
- Second interactor
- TSWV
Orthotospovirus tomatomaculaee0pathogen - Gene
- 9-lipoxygenase (LOX), 13-LOX, or α-dioxygenase-1 (α-DOX-1)e0
- Outcome
- programmed cell death (PCD)-associated symptomse0
- Direction
- attenuatede0
Not reportedMaterialCompoundOrgan / stageComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e5“in Nicotiana benthamiana”
e0“Silencing either 9-lipoxygenase (LOX), 13-LOX, or α-dioxygenase-1 (α-DOX-1) attenuated the programmed cell death (PCD)-associated symptoms caused by infection with either PVX-PVY or TSWV”
Title
Alberto García-Marcos; Remedios Pacheco; Aranzazu Manzano; et al. Journal of Virology 2013. doi:10.1128/jvi.03573-12
Other claims from this paper (5)
- Intervention effect Silencing COI1 expedited cell death during infection with compatible viruses.
- Association Enhanced expression of oxylipin biosynthesis genes and dioxygenase activity correlated with PVX-PVY infection.
- Intervention effect The Arabidopsis thaliana double lox1 α-dox-1 mutant became less susceptible to TSWV infection.
- Observation Oxylipin metabolism does not restrain virus accumulation in planta.
- Mechanism Oxylipin metabolism is a critical component that positively regulates PCD during compatible plant-virus interactions.