Claim · model-written summary
Exogenous IAA rescued fiber growth impaired by glucose signaling inhibition.
- Taxon
- cotton
Gossypium hirsutumt0 - Treatment
- exogenous application of either IAA or BRe0
- Outcome
- fiber growthe1
- Direction
- rescuede0
- Comparator
- inhibiting glucose signalinge1
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Verbatim
e0“this could be rescued by exogenous application of either IAA or BR”
e1“inhibiting glucose signaling by suppressing hexokinase activity through N-acetylglucosamine impaired fiber growth”
t0“cotton”
Title
Auxin and brassinosteroid synergically regulate cotton fiber elongation
Xinyang Li; Zhiqiang Zhang; Zhongying Ren; et al. Journal of Experimental Botany 2025. doi:10.1093/jxb/eraf483
Other claims from this paper (13)
- Observation IAA levels increased during fiber development.
- Association IAA levels were higher in long-fiber varieties.
- Intervention effect IAA promoted fiber elongation in a dose-dependent manner in ovule culture.
- Mechanism IAA activated BR biosynthesis.
- Mechanism IAA activated the BR signaling pathway.
- Intervention effect IAA partially rescued fiber elongation under brassinazole-induced BR deficiency.
- Intervention effect IAA partially rescued fiber elongation in pag1 mutants.
- Intervention effect BR supplementation partially alleviated fiber inhibition from impaired IAA transport.
- Intervention effect BR supplementation partially alleviated fiber inhibition from IAA deficiency.
- Mechanism Neither IAA nor BR fully compensated for absence of the other in fiber elongation.
- Intervention effect Hexokinase suppression by N-acetylglucosamine impaired fiber growth.
- Intervention effect Exogenous BR rescued fiber growth impaired by glucose signaling inhibition.
- Mechanism Glucose was inferred to act upstream of IAA and BR in regulating cotton fiber elongation.