Claim · model-written summary
HSR515 suppression reduced conversion of benzyl benzoate to SA in N. benthamiana.
- Taxon
- N. benthamiana
Nicotiana benthamianae0 - Treatment
- VIGS-mediated suppression of HSR515e0
- Gene
- HSR515e0
- Outcome
- conversion of benzyl benzoate to SAe0
- Direction
- reducede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“VIGS-mediated suppression of HSR515 in N. benthamiana reduced the conversion of benzyl benzoate to SA”
Title
Dawei Ma; Harley Gordon; Rashid Nazir; et al. The Plant Cell 2025. doi:10.1093/plcell/koaf241
Other claims from this paper (10)
- Intervention effect Benzaldehyde synthase knockout poplars produced less benzyl benzoate.
- Intervention effect Benzaldehyde synthase knockout poplars produced less benzyl salicylate.
- Intervention effect Benzaldehyde synthase knockout poplars produced less SA.
- Intervention effect Benzaldehyde synthase knockout poplars accumulated more benzoic acid.
- Mechanism HSR203J encodes a carboxylesterase that specifically hydrolyzes benzyl salicylate.
- Intervention effect HSR203J silencing reduced benzyl salicylate hydrolysis to SA in Nicotiana benthamiana.
- Mechanism The proposed pathway esterifies benzoyl-CoA to benzyl benzoate, converts it to benzyl salicylate, then releases SA.
- Observation HSR515 was identified as a pathogen-induced cytochrome P450 and putative benzyl benzoate 2-hydroxylase.
- Observation Brassicaceae genomes lacked HSR203J and HSR515 orthologs.
- Observation Other vascular plants contained HSR203J and HSR515 genes.