Claim · model-written summary
Disrupting SGT1 compromised GH7-triggered cell death.
- Taxon
- Nicotiana benthamiana
Nicotiana benthamianae0 - Treatment
- Genetic disruptione0
- Gene
- Suppressor of G2 allele of Skp1 (SGT1)e0
- Outcome
- GH7-triggered cell deathe0
- Direction
- compromisede0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“Genetic disruption of Nicotiana benthamiana Suppressor of G2 allele of Skp1 (SGT1) and Non-race specific Disease Resistance 1 (NDR1), but not Enhanced Disease Susceptibility 1 (EDS1), compromised GH7-triggered cell death”
Title
Xiao‐Bin Ji; Quan‐Le Zhang; Xin‐Xin Liu; et al. Plant Cell & Environment 2025. doi:10.1111/pce.70303
Other claims from this paper (8)
- Intervention effect Deleting each of six GH7 genes impaired Verticillium virulence.
- Intervention effect Deleting each GH7 gene did not affect fungal growth.
- Observation Each GH7 protein was confirmed to be secreted.
- Observation All GH7 proteins entered plant cells and localized in the cytoplasm.
- Observation GH7 proteins triggered hypersensitive cell death, reactive oxygen species burst and callose deposition independently of enzymatic activity.
- Intervention effect Disrupting NDR1 compromised GH7-triggered cell death.
- Intervention effect Disrupting EDS1 did not compromise GH7-triggered cell death.
- Mechanism The authors infer that GH7-triggered cell death partly depends on the SGT1-NDR1 pathway.