Claim · model-written summary
The LCF 12-hydroxyjasmonic acid glucoside induced leaf-folding in Samanea saman.
- Taxon
- Samanea saman
Samanea samane2 - Treatment
- LCF 12-hydroxyjasmonic acid glucosidee2
- Outcome
- leaf-foldinge2
- Direction
- inducese2
Not reportedComparator
Verbatim
e2“In Samanea saman, a model for nyctinasty, the LCF 12-hydroxyjasmonic acid glucoside induces leaf-folding by selectively targeting extensor motor cells and activating reactive oxygen species (ROS)-dependent K+ efflux via the SPORK2 channel”
Title
Bioorganic studies on the nyctinastic leaf-movement of plants
Minoru UEDA; Yuki MURAOKA Proceedings of the Japan Academy Series B 2026. doi:10.2183/pjab.102.002
Other claims from this paper (7)
- Mechanism Genus-specific leaf-closing and leaf-opening factors were identified as controlling the rhythmic leaf movement of legumes.
- Observation The LCF 12-hydroxyjasmonic acid glucoside selectively targets extensor motor cells in Samanea saman.
- Mechanism The LCF 12-hydroxyjasmonic acid glucoside activates reactive oxygen species (ROS)-dependent K+ efflux via the SPORK2 channel in Samanea saman.
- Mechanism SPORK2 and the SsSLAH1/SsSLAH3 anion channel complex are key effectors in regulating leaf movement in Samanea saman.
- Intervention effect Low temperatures inactivate SPORK2, which functions as a temperature-sensitive K+ channel in Samanea saman.
- Mechanism SPORK2 mediates rain-induced leaf folding in Samanea saman.
- Mechanism Plant K+ channels have a previously unrecognized role in temperature sensing.
